鞘脂
神经酰胺
内科学
内分泌学
胆固醇
免疫印迹
胆囊
p38丝裂原活化蛋白激酶
信号转导
鞘糖脂
磷酸化
MAPK/ERK通路
生物
生物化学
医学
细胞凋亡
基因
作者
Beom Jae Lee,Jae J. Kim,Byung Kyu Kim,Sung Joo Jung,Moon Kyung Joo,Seung Goun Hong,Jang Soo Kim,Ji Hoon Kim,Jong Eun Yeon,Jong‐Jae Park,Kwan Soo Byun,Young‐Tae Bak,Hwan‐Soo Yoo,Seikwan Oh,Young‐Tae Bak,Hwan‐Soo Yoo,Seikwan Oh
标识
DOI:10.1111/j.1440-1746.2010.06246.x
摘要
Abstract Background: Sphingolipids play a very important role in cell membrane formation, signal transduction and plasma lipoprotein metabolism. The first rate‐limiting step in the sphingolipid biosynthetic pathway is catalyzed by serine palmitoyltransferase (SPT), and myriocin is a potent and specific inhibitor of SPT. We investigated the impact of SPT inhibition on cholesterol gallstone formation in C57BL/6J mice. Methods: Three groups of eight‐week‐old C57BL/6J mice were utilized. Each group consisted of 20 mice; group A, B, and C were fed normal chow, lithogenic diet with phosphate buffered saline, and lithogenic diet with myriocin (0.3 mg/kg), respectively, for 6 weeks. The ceramide levels in both serum and bile were assessed by high performance liquid chromatography analysis. Protein expression of ERK, JNK and p38 in the extracted gallbladder were determined by Western‐blot analysis. Results: Myriocin treatment caused a significant decrease in the rate of cholesterol gallstone formation. The lithogenic diet mice (group B) showed the highest ceramide activities in both the serum and bile among all the tested groups and there was significant suppression of the ceramide levels in both the serum and bile of the myriocin‐treated mice (group C, p < 0.05). Phosphorylation of p38 in the gallbladder was increased in the lithogenic‐diet mice and the expression of phosphorylated p38 was significantly suppressed in the myriocin treated mice. Conclusions: SPT inhibition by myriocin suppressed gallstone formation and the levels of ceramide in both the serum and bile. p38 in the cellular signaling pathways might be associated with cholesterol gallstone formation.
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