MIF inhibition alleviates vitiligo progression by suppressing CD8+ T cell activation and proliferation

白癜风 CD8型 免疫学 巨噬细胞移动抑制因子 细胞毒性T细胞 细胞因子 T细胞 癌症研究 免疫系统 医学 生物 体外 生物化学
作者
Jianru Chen,Weinan Guo,Pengran Du,Tingting Cui,Yuqi Yang,Yinghan Wang,Pan Kang,Zhe Zhang,Qi Wang,Zhubiao Ye,Ling Liu,Zhe Jian,Tianwen Gao,Huijie Bian,Shuli Li,Chunying Li
出处
期刊: 卷期号:260 (1): 84-96 被引量:57
标识
DOI:10.1002/path.6073
摘要

Abstract In vitiligo, autoreactive CD8 + T cells have been established as the main culprit considering its pathogenic role in mediating epidermal melanocyte‐specific destruction. Macrophage migration inhibitory factor (MIF) is a pleiotropic molecule that plays a central role in various immune processes including the activation and proliferation of T cells; but whether MIF is intertwined in vitiligo development and progression and its involvement in aberrantly activated CD8 + T cells remains ill‐defined. In this study, we found that MIF was overabundant in vitiligo patients and a mouse model for human vitiligo. Additionally, inhibiting MIF ameliorated the disease progression in vitiligo mice, which manifested as less infiltration of CD8 + T cells and more retention of epidermal melanocytes in the tail skin. More importantly, in vitro experiments indicated that MIF‐inhibition suppressed the activation and proliferation of CD8 + T cells from the lymph nodes of vitiligo mice, and the effect extended to CD8 + T cells in peripheral blood mononuclear cells of vitiligo patients. Finally, CD8 + T cells derived from MIF‐inhibited vitiligo mice also exhibited an impaired capacity for activation and proliferation. Taken together, our results show that MIF might be clinically targetable in vitiligo treatment, and its inhibition might ameliorate vitiligo progression by suppressing autoreactive CD8 + T cell activation and proliferation. © 2023 The Pathological Society of Great Britain and Ireland.
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