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Abstract 6369: Perivascular niche-resident alveolar macrophages confer interstitial pneumonitis related to trastuzumab deruxtecan

曲妥珠单抗 病理 肺泡巨噬细胞 牙槽壁 巨噬细胞 医学 利基 内科学 化学 癌症 乳腺癌 体外 生物化学
作者
Qing Wei,Teng Yang,Ziwen Zhang,Jiayu Zhu,Xu Xue,Jieer Ying,Peng Guo,Xiangdong Cheng
出处
期刊:Cancer Research [American Association for Cancer Research]
卷期号:85 (8_Supplement_1): 6369-6369
标识
DOI:10.1158/1538-7445.am2025-6369
摘要

Abstract Background: T-DXd, a HER2-targeting antibody-drug conjugate (ADC), has shown transformative potential in cancer therapy but is implicated in causing fatal interstitial lung disease (ILD) in clinical trials. The mechanistic basis for these ADC-induced adverse effects remains unclear. This study aimed to investigate whether T-DXd-induced ILD represents an off-target adverse event (AE) and to explore the underlying cellular and molecular mechanisms. Methods: We analyzed HER2 antigen expression in a panel of 98 normal human lung tissues to determine whether T-DXd-induced ILD is on-target or off-target. To study the mechanisms of ILD, we developed an immunocompetent murine model that replicates T-DXd-induced lung toxicity. Using single-cell RNA sequencing, we identified alveolar macrophages (AMs) as the primary cell type affected by T-DXd in the lung microenvironment. Intravital microscopy was employed to visualize how perivascular AMs engulf T-DXd via Fc-FcγR engagement. To mitigate T-DXd-induced toxicity, a pre-conditioning strategy using IgG1 or the parental antibody of T-DXd was tested in the murine model. Results: Analysis of HER2 expression in normal lung tissues revealed minimal expression, confirming that T-DXd-induced ILD is an off-target AE. Single-cell RNA sequencing and intravital microscopy identified perivascular AMs as the key mediators of lung toxicity, driven by Fc-FcγR engagement with T-DXd. This interaction triggered a phenotypic shift in AMs toward an inflammatory, pro-ILD state, characterized by activation of the SPP1 pathway. Pre-conditioning AMs with IgG1 or the parental antibody of T-DXd effectively reduced ADC uptake, alleviating lung inflammation and immune activation in the murine model. Conclusion: T-DXd-induced ILD is an off-target AE mediated by Fc-FcγR-driven phagocytosis of T-DXd by perivascular alveolar macrophages. This interaction promotes an inflammatory SPP1high phenotype in AMs, leading to lung inflammation and immune activation. Pre-conditioning strategies targeting AMs offer a potential clinical approach to reduce nonspecific ADC uptake and mitigate lung toxicity. These findings provide significant insights into the mechanisms of ADC-induced toxicities and potential intervention strategies. Citation Format: Qing Wei, Teng Yang, Ziwen Zhang, Jiayu Zhu, Xiru Xue, Jieer Ying, Peng Guo, Xiangdong Cheng. Perivascular niche-resident alveolar macrophages confer interstitial pneumonitis related to trastuzumab deruxtecan [abstract]. In: Proceedings of the American Association for Cancer Research Annual Meeting 2025; Part 1 (Regular Abstracts); 2025 Apr 25-30; Chicago, IL. Philadelphia (PA): AACR; Cancer Res 2025;85(8_Suppl_1):Abstract nr 6369.

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