后脑区
神经降压素
加压素
内分泌学
内科学
阿扑吗啡
蛙皮素
血管紧张素II
神经肽
催产素
血管紧张素Ⅲ
胆囊收缩素
化学
多巴胺能
多巴胺
受体
医学
血管紧张素Ⅱ受体1型
作者
Michael Wu,R. K. Harding,Herman Hugenholtz,John Kucharczyk
出处
期刊:Peptides
[Elsevier]
日期:1985-01-01
卷期号:6: 173-175
被引量:24
标识
DOI:10.1016/0196-9781(85)90028-2
摘要
Short-latency emetic responses were induced in dogs by injecting angiotensin II (AII), arginine vasopressin (AVP), and neurotensin (NTN) into cerebroventricular (ICV) and cisternal (ICT) sites also responsive to the emetic effects of apomorphine (APO). Angiotensin III, bradykinin, bombesin, oxytocin, adrenocorticotropic hormone, substance P, gastrin-related peptide and cholecystokinin were ineffective. The results suggest a possible dopaminergic mediation of peptide-induced emesis by receptors in the area postrema (AP).
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