基因敲除
胶质母细胞瘤
MAPK/ERK通路
癌症研究
信号转导
p38丝裂原活化蛋白激酶
医学
细胞生物学
生物
细胞凋亡
遗传学
作者
Wansoo Kim,Song Park,Seung Hyun Han,Hee-Yeon Kim,Seoung-Woo Lee,Dae‐Hwan Kim,Soyoung Jang,Jin‐Kyu Park,Jee Eun Han,Choonok Kim,Junho Cho,Ethan Seah,Jiyeon Lee,Zae Young Ryoo,Seong‐Kyoon Choi
出处
期刊：Anticancer Research
[International Institute of Anticancer Research (IIAR) Conferences 1997. Athens, Greece. Abstracts]
日期：2025-01-31
卷期号：45 (2): 549-564
被引量：2
标识
DOI：10.21873/anticanres.17443
摘要
PRPF4 knockdown inhibits GBM progression by reducing p38 MAPK and ERK signaling cascade with metabolic alterations. Targeting PRPF4 may offer novel therapeutic strategies for GBM treatment.
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