炎症
败血症
凝结
医学
急性肾损伤
生长因子
免疫学
内科学
受体
作者
Hongwei Wang,Minmin Wu,Mian-Mian Zhu,Yiyang Qin,Keqi Wang,Chenyu Wu,Rongrong Zhang,Yin Wang,Chen Zhou,Shuang Luo,Chaosheng Lu,Jingye Pan
标识
DOI:10.1186/s11658-025-00762-2
摘要
In summary, these findings indicate that GDF11 is a potential therapeutic approach for SAKI and highlight the crucial role of PGC-1α/Nrf2 signaling in GDF11-mediated renal protection during SAKI.
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