炎症体
医学
心脏病学
内科学
信号转导
细胞生物学
心房颤动
炎症
生物
作者
Chunxia Yao,Tina Veleva,Larry Scott,Shuyi Cao,Luge Li,Gong Chen,Prince Jeyabal,Xiaolu Pan,Katherina M. Alsina,Issam Abu-Taha,Shokoufeh Ghezelbash,Corey Reynolds,Ying H. Shen,Scott A. LeMaire,Wilhelm Schmitz,Frank U. Müller,Ali El‐Armouche,N. Tony Eissa,Christine Beeton,Stanley Nattel
出处
期刊：Circulation
[Ovid Technologies (Wolters Kluwer)]
日期：2018-05-25
卷期号：138 (20): 2227-2242
被引量：561
标识
DOI：10.1161/circulationaha.118.035202
摘要
Atrial fibrillation (AF) is frequently associated with enhanced inflammatory response. The NLRP3 (NACHT, LRR, and PYD domain containing protein 3) inflammasome mediates caspase-1 activation and interleukin-1β release in immune cells but is not known to play a role in cardiomyocytes (CMs). Here, we assessed the role of CM NLRP3 inflammasome in AF.
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