Lv2
170 积分 2024-08-30 加入
Disulfidptosis in cancer: from redox stress to therapeutic strategy
1个月前
已完结
Cell-death networks
2个月前
已完结
Fluorescent AIE-Active Materials for Two-Photon Bioimaging Applications
5个月前
已完结
Quantitation of calreticulin exposure associated with immunogenic cell death
6个月前
已完结
Fullerenols boosting the therapeutic effect of anti-CD47 antibody to trigger robust anti-tumor immunity by inducing calreticulin exposure
6个月前
已完结
Calreticulin exposure induced by anticancer drugs is associated with the p53 signaling pathway in colorectal cancer cells
6个月前
已完结
An Endoplasmic Reticulum Stress‐Specific Nanoinducer Selectively Evokes Type‐II Immunogenic Cell Death for Pyroptotic Cancer Immunotherapy
6个月前
已完结
Nanomedicines for an Enhanced Immunogenic Cell Death-Based In Situ Cancer Vaccination Response
6个月前
已完结
Calreticulin exposure dictates the immunogenicity of cancer cell death
6个月前
已完结
The Next Decade of Immune Checkpoint Therapy
7个月前
已完结
PCK1 deficiency promotes MASH-HCC progression by 12-HETE-induced CD8 + T cell dysfunction
6个月前
已采纳
PCK1 deficiency promotes MASH-HCC progression by 12-HETE-induced CD8 + T cell dysfunction
6个月前
已采纳
PCK1 deficiency promotes MASH-HCC progression by 12-HETE-induced CD8 + T cell dysfunction
6个月前
已采纳
PCK1 deficiency promotes MASH-HCC progression by 12-HETE-induced CD8 + T cell dysfunction
6个月前
已采纳
PCK1 deficiency promotes MASH-HCC progression by 12-HETE-induced CD8 + T cell dysfunction
6个月前
已采纳
PCK1 deficiency promotes MASH-HCC progression by 12-HETE-induced CD8+ T cell dysfunction
6个月前
已采纳
PCK1 deficiency promotes MASH-HCC progression by 12-HETE-induced CD8 + T cell dysfunction
6个月前
已采纳
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