哈卡特
蛋白激酶B
PI3K/AKT/mTOR通路
下调和上调
癌症研究
体内
特应性皮炎
角质形成细胞
信号转导
免疫学
生物
医学
体外
细胞生物学
生物化学
生物技术
基因
作者
Maoxin Huang,Liping Dong,Yue He,Xinying Cai,Yun Lü,Jingjing Xu,Feng‐Li Xiao
标识
DOI:10.1016/j.alit.2025.04.005
摘要
This study identifies YTHDF1 as a contributor to AD pathogenesis by influencing keratinocyte proliferation and differentiation. It also suggests that YTHDF1 could be a potential therapeutic target for AD treatment.
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