The role of ADAMTS13 in acute myocardial infarction: cause or consequence?
心脏病学
心肌梗塞
医学
内科学
作者
Elise S. Eerenberg,Paul F. Teunissen,Bert‐Jan H. van den Born,Joost C.M. Meijers,Maurits R. Hollander,Matthijs Jansen,R. Tijssen,Jeroen A.M. Beliën,Peter M. van de Ven,M. F. A. Aly,Otto Kamp,Hans W.M. Niessen,Pieter W. Kamphuisen,Marcel Levi,Niels van Royen
出处
期刊：Cardiovascular Research [Oxford University Press] 日期：2016-05-12卷期号：111 (3): 194-203被引量：25
No correlation was found between VWF/ADAMTS13 and infarct size in patients. However, patients suffering from IMH had significantly higher VWF activity and lower ADAMTS13 activity. Intracoronary administration of rADAMTS13 did not decrease infarct size or IMH in a porcine model of myocardial ischaemia-reperfusion. These data dispute the imbalance in ADAMTS13 and VWF as the cause of no reflow.
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