The Anti-Inflammatory Peptide TnP Is a Candidate Molecule for Asthma Treatment

免疫学 医学 哮喘 肺 炎症 免疫球蛋白E 恶化 支气管高反应性 哮喘的病理生理学 增生 呼吸道疾病 病理 内科学 抗体
作者
Carla Lima,Maria Alice Pimentel Falcão,Felipe Justiniano Pinto,Jefferson Thiago Gonçalves Bernardo,Mônica Lopes‐Ferreira
出处
期刊:Cells [Multidisciplinary Digital Publishing Institute]
卷期号:12 (6): 924-924 被引量:6
标识
DOI:10.3390/cells12060924
摘要

Asthma is the most common chronic lung disease, with increasing morbidity and mortality worldwide. Accumulation of peribronchial leukocytes is the hallmark of asthma, in particular, eosinophils, which have been reported as the primary cell associated with the induction of airway hyperresponsiveness. Continued exacerbation and accumulation of other leukocytes, such as neutrophils, Th1, and Th17 cells correlate with many of the long-term effects of asthma, such as airway remodeling. We have patented the TnP family of synthetic cyclic peptides, which is in the preclinical phase of developmental studies for chronic inflammatory diseases. The aim of this work was to investigate whether TnP could show anti-inflammatory activity in a murine model of asthma that includes a mixed phenotype of eosinophilic and neutrophilic inflammation. For this, Balb/c mice, sensitized with OVA and exposed to 1% challenge with OVA aerosol, were submitted to prophylactic treatment, receiving TnP at 0.3 mg/kg orally, 1 h before each challenge. We found that sensitized mice challenged with OVA and treated with TnP showed no airway hyperreactivity or lung remodeling. TnP acts systemically in secondary lymphoid organs and locally in the lung, inhibiting the production of Th2/Th17 cytokines. Furthermore, TnP prevented the infiltration of eosinophils and neutrophils in the BAL and lung tissue, inhibited the production of IgE/IgG1, prevented hyperplasia of mucus-producing cells, and decreased the thickening and deposition of sub-epithelial collagen. Our results showed TnP as a candidate molecule for the treatment of airway remodeling associated with inflammatory diseases, such as asthma.
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