细胞生物学
细胞毒性T细胞
CD16
受体
生物
刺激
淋巴细胞功能相关抗原1
免疫突触
白细胞介素21
神经细胞粘附分子
自然杀伤细胞
T细胞
细胞因子
细胞粘附分子
细胞粘附
CD8型
细胞
免疫学
T细胞受体
免疫系统
细胞间粘附分子-1
体外
生物化学
内分泌学
CD3型
作者
Doris Urlaub,Kristine Höfer,Martha‐Lena Müller,Carsten Watzl
出处
期刊:Journal of Immunology
[American Association of Immunologists]
日期:2017-01-19
卷期号:198 (5): 1944-1951
被引量:91
标识
DOI:10.4049/jimmunol.1601004
摘要
Abstract The integrin LFA-1 is essential for efficient activation and for cytotoxicity of NK cells because it initiates the assembly of the immunological synapse and mediates firm adhesion to the target. LFA-1 is also needed to polarize the cytotoxic machinery of the NK cell toward the target cell. The binding affinity and avidity of integrins can be regulated via inside-out signals from other receptors. In this article, we investigate the signals necessary to activate LFA-1 in human NK cells. Our data show that LFA-1 has a low ligand-binding activity in resting human NK cells, but it can be stimulated by triggering activating receptors, such as 2B4 or CD16, or by coactivation of different receptor combinations. Short-term stimulation of freshly isolated NK cells with cytokines, such as IL-15, IL-12, or IL-18, does not activate LFA-1 but increases the responsiveness of the cells to subsequent receptor stimulation. Different NK cell subsets vary in their ability to induce LFA-1 binding activity after activating receptor stimulation. Interestingly, the NK cell subsets that are more mature and possess higher cytotoxic potential also show the highest activation of LFA-1, which correlated with the expression of the small calcium-binding protein S100A4. Our data suggest that regulation of LFA-1 is one reason for the different activity of NK cells during differentiation.
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