内质网
细胞生物学
内吞循环
抗原呈递
内体
抗原处理
抗原
肽
MHC I级
舱室(船)
化学
胞浆
内吞作用
细胞内
小泡
生物物理学
主要组织相容性复合体
转运蛋白
过程(计算)
溶酶体
生物
细胞内转运
与抗原处理相关的转运体
生物化学
细胞质
调节器
作者
J. Luke Postoak,Alyssa Koch,Sicong Shan,Ray A Ohara,Suin Jo,Jing Chen,Pritesh Desai,Feiya Ou,Magdalena Kraft,Michael S. Diamond,Sunkyung Kim,Theresa L Murphy,Kenneth M. Murphy,J. Luke Postoak,Alyssa Koch,Sicong Shan,Ray A Ohara,Suin Jo,Jing Chen,Pritesh Desai
标识
DOI:10.1073/pnas.2519922122
摘要
The intracellular processing route used by type I conventional dendritic cells (cDC1) for cross-presentation of viral- or tumor-associated antigens remains controversial. One model proposes that captured antigens exit from damaged phagolysosomes and enter the cytosol, where they are processed for presentation by MHC class I molecules (MHC-I). This model relies on proteasomal degradation and TAP-dependent peptide transport into the endoplasmic reticulum (ER) for loading by the peptide loading complex (PLC). An alternative model proposes a vesicular route in which captured antigens are retained and processed within an endocytic compartment. A compelling argument favoring the cytosolic model is the dramatic loss of cross-presentation by TAP-deficient cDC1, which presumes that peptides derived from phagocytosed cells require TAP for their transport into the ER to reach the PLC. However, here we show that cross-presentation by cDC1 is TAP-dependent because TAP is required for the normal trafficking of MHC-I molecules. Our work demonstrates that MHC-I must reach a post-Golgi compartment for loading in cross-presentation and that this process may occur independently of TAP.
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