GNAS复合轨迹
乳腺癌
医学
癌症研究
蛋白激酶B
细胞生长
PI3K/AKT/mTOR通路
基因沉默
癌症
肿瘤科
内科学
信号转导
生物
细胞生物学
遗传学
基因
作者
Xiaosong Jin,Li Zhu,Zhuhong Cui,Jing Tang,Mingjun Xie,Guosheng Ren
标识
DOI：10.1007/s12094-019-02042-w
摘要
The data suggested that GNAS promoted breast cancer cell proliferation and migration (EMT) through the PI3K/AKT/Snail1/E-cadherin signaling pathway. These findings also indicate that GNAS can serve as a potential prognostic indicator and novel therapeutic target in breast cancer.
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(2025-6-4)
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