氧化应激
药理学
活性氧
超氧化物歧化酶
创伤性脑损伤
吴茱萸碱
医学
小干扰RNA
细胞凋亡
化学
内科学
生物化学
转染
精神科
基因
作者
Min Xu,Wenhua Wang,Wei Lu,Xiaoyang Ling,Rui Qin,Haibo Ni
标识
DOI:10.1016/j.biopha.2022.113435
摘要
Taken together, we demonstrated that Evo improved the outcomes after TBI by targeting the PGK1/NRF2 signaling-regulated oxidative stress. Evo may represent a potential therapy to promote recovery from TBI.
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