细胞生物学
巨噬细胞极化
心肌细胞
Wnt信号通路
小RNA
炎症
微泡
生物
癌症研究
化学
信号转导
巨噬细胞
免疫学
体外
生物化学
基因
作者
Zhiwen Luo,Beijie Qi,Yaying Sun,Yisheng Chen,Jinrong Lin,Haocheng Qin,Ning Wang,Runjie Shi,Xiliang Shang,Shiyi Chen,Jiwu Chen
出处
期刊:Small
[Wiley]
日期:2022-07-08
卷期号:18 (34)
被引量:65
标识
DOI:10.1002/smll.202201957
摘要
Severe inflammation and myogenic differentiation disorder are the major obstacles to skeletal muscle healing after injury. MicroRNAs (miRNAs) play an important role as regulatory molecules during the process of muscle healing, but the detailed mechanism of miRNA-mediated intercellular communication between myoblasts and macrophages remains unclear. Here, it is reported that myoblasts secrete miRNAs-enriched exosomes in the inflammatory environment, through which miR-224 is transferred into macrophages to inhibit M2 polarization. Further data demonstrate that WNT-9a may be a direct target of miR-224 for macrophage polarization. In turn, the secretome of M1 macrophages impairs myogenic differentiation and promotes proliferation. Single-cell integration analysis suggests that the elevation of exosome-derived miR-224 is caused by the activation of the key factor E2F1 in myoblasts and demonstrates the RB/E2F1/miR-224/WNT-9a axis. In vivo results show that treatment with antagomir-224 or liposomes containing miR-224 inhibitors suppresses fibrosis and improves muscle recovery. These findings indicate the importance of the crosstalk between myoblasts and macrophages via miRNA-containing exosomes in the regulation of macrophage polarization and myogenic differentiation/proliferation during muscle healing. This study provides a strategy for treating muscle injury through designing an M2 polarization-enabling anti-inflammatory and miRNA-based bioactive material.
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