医学
协同失调
逼尿肌括约肌协同失调
排尿困难
泌尿系统
上尿路
膀胱输尿管反流
膀胱过度活动
泌尿科
神经源性膀胱功能障碍
尿失禁
泌尿系疾病
下尿路症状
膀胱
外科
内科学
回流
神经系统疾病
中枢神经系统疾病
病理
前列腺
替代医学
疾病
癌症
作者
Zhonghan Zhou,Yi Gao,Xing Li,Xuesheng Wang,Limin Liao
摘要
Abstract Purpose To review the characteristics of the neurogenic lower urinary tract dysfunction (NLUTD) secondary to aortic dissection (AD), analyze the clinical features, and discuss the treatment options. Methods Ten individuals complaining of lower urinary tract syndrome following AD were enrolled in this study. Clinical characteristics, urological and neurological symptoms/signs, imaging examination, and intervention were reviewed. Liao's comprehensive classification system was used to precisely assess the lower and upper urinary dysfunction. Results The urinary symptoms can be varied, including dysuria, incontinence, and frequency. Individuals were divided into the detrusor overactivity (DO) and detrusor underactivity (DU) subgroups. Continence, impaired upper urinary tract functions (renal insufficiency, vesicoureteral reflux, upper urinary tract dilatation, and lower urinary tract functions (DO and/or detrusor external sphincter dyssynergia/detrusor bladder neck dyssynergia, DU, low bladder capacity, and compliance) were examined using video‐urodynamics. The principle of treatment is “low‐pressure bladder storage with complete bladder emptying,” and close follow‐up was recommended due to the volatile course of NLUTD. Conclusions Both cardiovascular surgeons and urologists should pay attention to the occurrence of NLUTD following AD, and determine the most appropriate therapeutic option.
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