生物
免疫学
白细胞介素12
白细胞介素21
CD8型
细胞毒性T细胞
PI3K/AKT/mTOR通路
肿瘤坏死因子α
蛋白激酶B
干扰素
细胞因子
癌症研究
细胞生物学
信号转导
免疫系统
生物化学
体外
作者
Zhidong Hu,Hui-Min Zhao,Minjing Li,Xuhui Liu,Daniel Barkan,Douglas B. Lowrie,Shuihua Lu,Xiao‐Yong Fan
标识
DOI：10.1093/infdis/jiy046
摘要
Our study delineated the profile of KLRG1+CD4+ T cells in patients with tuberculosis and suggests that M. tuberculosis infection drives CD4+ T cells to acquire increased effector function in a terminally differentiated state, which is restrained by KLRG1 via KLRG1/Akt signaling pathway.
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