促性腺激素
正中隆起
内分泌学
内科学
移植
人绒毛膜促性腺激素
激素
促性腺激素释放激素
生物
排卵
医学
促黄体激素
作者
Ann-Judith Silverman,M.E. Gibson
标识
DOI:10.1016/1043-2760(90)90100-h
摘要
The defect of the hypogonadal mouse, resulting in infantile reproductive organs and severely reduced gonadotropin levels, is due to a truncation of the gene encoding for preprogonadotropinreleasing hormone. The hypogonadal mouse bearing a graft containing normal gonadotropin-releasing hormone neurons may show testicular development, seminal vesicle growth, and increased gonadotropin production in males. Normalization of gonadotropin levels in females is frequently associated with the capacity for a reflex ovulation followed by pregnancy and bearing of live young. All of these phenomena are dependent on the outgrowth of gonadotropin-releasing hormone axons from the graft to the host median eminence and the hypophysial portal capillaries.
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