Green tea polyphenol EGCG reverse cisplatin resistance of A549/DDP cell line through candidate genes demethylation

生物 顺铂 A549电池 细胞凋亡 去甲基化 细胞周期 分子生物学 细胞生长 癌症研究 基因表达 DNA甲基化 生物化学 基因 遗传学 化疗
作者
Youwei Zhang,Xiang Wang,Liang Han,Yizhou Zhou,Sanyuan Sun
出处
期刊:Biomedicine & Pharmacotherapy [Elsevier BV]
卷期号:69: 285-290 被引量:76
标识
DOI:10.1016/j.biopha.2014.12.016
摘要

Epigallocatechin-3-gallate (EGCG), the major polyphenol in green tea, has been extensively studied as a potential demethylating agent. Our hypothesis is that EGCG could resensitize non-small-cell lung cancer (NSCLC) cells to cisplatin (DDP) through candidate genes demethylation. The A549/DDP cell line was established by continuous exposure of A549 cells to increasing concentrations of DDP. MTT, colony formation assay, flow cytometric analysis, Hoechst staining, real time-PCR, quantitative methylation-specific PCR and in vivo experiments were performed in this study. EGCG+DDP treatment significantly caused proliferation inhibition, cell cycle arrest in G1 phase, increase of apoptosis in A549/DDP cells, along with inhibition of DNA methyltransferase (DNMT) activity and histone deacetylase (HDAC) activity, reversal of hypermethylated status and downregulated expression of GAS1, TIMP4, ICAM1 and WISP2 gene in A549/DDP cells. Furthermore, pre-treatment with EGCG followed by DDP caused significant tumor inhibition in vivo. Methylation levels of GAS1, TIMP4, ICAM1 and WISP2 were decreased and their expression levels were increased in EGCG-treatment groups, but only combinatorial treatment group caused growth inhibition. In conclusion, we identified EGCG pretreatment resensitized cells to DDP, along with the demethylation and restoration of expression of candidate genes.
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