PTPN11型
神经母细胞瘤RAS病毒癌基因同源物
克拉斯
髓系白血病
临床意义
医学
癌症研究
内科学
肿瘤科
生物
癌症
结直肠癌
作者
Taeko Kaburagi,Genki Yamato,Norio Shiba,Kenichi Yoshida,Yusuke Hara,Ken Tabuchi,Yuichi Shiraishi,Kentaro Ohki,Manabu Sotomatsu,Hirokazu Arakawa,H. Matsuo,Akira Shimada,Tomohiko Taki,Nobutaka Kiyokawa,Daisuke Tomizawa,Keizo Horibe,Satoru Miyano,Takashi Taga,Souichi Adachi,Seishi Ogawa,Yasuhide Hayashi
出处
期刊:Haematologica
[Ferrata Storti Foundation]
日期:2021-03-18
卷期号:107 (3): 583-592
被引量:12
标识
DOI:10.3324/haematol.2020.269431
摘要
RAS pathway alterations have been implicated in the pathogenesis of various hematological malignancies. However, their clinical relevance in pediatric acute myeloid leukemia (AML) is not well characterized. We analyzed the frequency, clinical significance, and prognostic relevance of RAS pathway alterations in 328 pediatric patients with de novo AML. RAS pathway alterations were detected in 80 (24.4%) of 328 patients: NF1 (n=7, 2.1%), PTPN11 (n=15, 4.6%), CBL (n=6, 1.8%), NRAS (n=44, 13.4%), KRAS (n=12, 3.7%). Most of these alterations in the RAS pathway were mutually exclusive also together with other aberrations of signal transduction pathways such as FLT3-ITD (P=0.001) and KIT mutation (P=0.004). NF1 alterations were frequently detected in patients with complex karyotype (P=0.031) and were found to be independent predictors of poor overall survival (OS) in multivariate analysis (P=0.007). At least four of seven patients with NF1 alterations had biallelic inactivation. NRAS mutations were frequently observed in patients with CBFB-MYH11 and were independent predictors of favorable outcomes in multivariate analysis (OS, P=0.023; event-free survival [EFS], P=0.037). Patients with PTPN11 mutations more frequently received stem cell transplantation (P=0.035) and showed poor EFS than patients without PTPN11 mutations (P=0.013). Detailed analysis of RAS pathway alterations may enable a more accurate prognostic stratification of pediatric AML and may provide novel therapeutic molecular targets related to this signal transduction pathway.
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