A Recombinant Oncolytic Influenza Virus Carrying GV1001 Triggers an Antitumor Immune Response

溶瘤病毒 病毒 生物 病毒学 免疫系统 重组DNA 免疫疗法 重组病毒 甲型流感病毒 细胞毒性T细胞 体外 免疫学 基因 生物化学
作者
Cong Li,Yuying Tian,Fang Sun,Guanglin Lei,Jinxia Cheng,Chongyu Tian,Hongyu Yu,Zhuoya Deng,Shuai Lü,Lishi Wang,Ruixue Xiao,Changqing Bai,Penghui Yang
出处
期刊:Human Gene Therapy [Mary Ann Liebert, Inc.]
卷期号:35 (1-2): 48-58 被引量:11
标识
DOI:10.1089/hum.2022.206
摘要

Oncolytic viruses are able to lyse tumor cells selectively in the liver without killing normal hepatocytes, in addition to activating the immune response. Oncolytic virus therapy is expected to revolutionize the treatment of liver cancer, including hepatocellular carcinoma (HCC), one of the most frequent and fatal malignancies. In this study, reverse genetics techniques were exploited to load NA fragments of the A/PuertoRico/8/34 virus (PR8) with GV1001 peptides derived from human telomerase reverse transcriptase. An in vitro assessment of the therapeutic effect of the recombinant oncolytic virus was followed by an in vivo study in mice with HCC. The recombinant virus was verified by sequencing of the recombinant viral gene sequence, and viral virulence was detected by hemagglutination assays and based on the 50% tissue culture infectious dose (TCID50). The morphological structure of the virus was observed by electron microscopy, and GV1001 peptide was localized by cellular immunofluorescence. The selective cytotoxicity of the recombinant oncolytic virus in vitro was demonstrated in cultured HCC cells and normal hepatocytes, as only the tumor cells were killed; the normal cells were not significantly altered. Consistent with the in vitro results, the recombinant oncolytic influenza virus significantly inhibited liver tumor growth in mice in vivo, in addition to inducing an antitumor immune response, including an increase in the number of CD4+ and CD8+ T lymphocytes and, in turn, improving survival. Our results suggest that oncolytic influenza virus carrying GV1001 is a promising immunotherapy in patients with HCC.
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