癌变
KEAP1型
癌症研究
糖酵解
细胞凋亡
化学
医学
生物化学
内科学
癌症
转录因子
新陈代谢
基因
作者
Liang Zhao,Min Soo Kang,Xiaomeng Liu,Zhenran Wang,Yan Wang,Chien‐Jen Chen,Wenhui Liu,Shiqian Liu,Baibei Li,Chong Li,Antao Chang,Bo Tang
标识
DOI：10.1186/s13046-022-02528-6
摘要
These results collectively establish UBR7 as a critical negative regulator of aerobic glycolysis and HCC tumorigenesis through regulation of the Keap1/Nrf2/Bach1/HK2 axis, providing a potential clinical and therapeutic target for the HCC treatment.
科研通智能强力驱动
Strongly Powered by AbleSci AI
PDF的下载单位、IP信息已删除
(2025-6-4)
huanhuanhuan
v0id
Yinxb
极限001
DW
璐璐
嘻嘻哈哈
学霸业
小马宝莉
aajhajkahna
Ray
乐空思
谢雷XIELei
lalala
invisight
蓝天
XYZONE
渡人舟
囧囧
uuu
皖公网安备34019202002308
