Endothelial Cell Phenotypic Plasticity in Cardiovascular Physiology and Disease: Mechanisms and Therapeutic Prospects

重编程 医学 内皮功能障碍 间充质干细胞 疾病 人口 再生医学 内皮干细胞 生物信息学 内皮 免疫学 神经科学 干细胞 细胞生物学 生物 病理 细胞 心脏病学 内科学 遗传学 体外 环境卫生
作者
Diego Barbosa de Queiroz,Juliana Montenegro Parente,Laena Pernomian,Emily Waigi,Mabruka Alfaidi,Wenbin Tan,Cameron G. McCarthy,Camilla F. Wenceslau
出处
期刊:American Journal of Hypertension [Oxford University Press]
卷期号:38 (7): 411-421 被引量:1
标识
DOI:10.1093/ajh/hpaf027
摘要

Abstract Endothelial cells (ECs) are a highly specialized and heterogeneous population that plays a fundamental role in maintaining vascular homeostasis, immune regulation, and blood flow control. Beyond serving as a physical barrier, ECs exhibit remarkable plasticity, undergoing phenotypic transitions, including endothelial-to-mesenchymal (EndMT), endothelial-to-hematopoietic (EndHT), endothelial-to-osteoblast (EndOT) and endothelial-to-immune-cell-like (EndICLT). These transitions allow ECs to adapt to developmental, physiological, and pathological conditions. Advances in single-cell RNA sequencing (scRNA-seq) and associated technologies have provided deeper insights into the molecular diversity of ECs across different vascular beds and stages of development, revealing their transcriptional heterogeneity and specialized functions. For example, ECs within the aortic arch display distinct phenotypic variations depending on their location, reflecting adaptations to regional differences in blood flow and shear stress. Activated EndMT has been implicated in the progression of various cardiovascular diseases, including hypertension, atherosclerosis, and vascular malformations by contributing to endothelial dysfunction, vascular wall inflammation, and remodeling. Recent therapeutic approaches aim to mitigate EndMT-associated vascular damage through interventions such as endothelial reprogramming, statins, and autophagy enhancers. Partial reprogramming of ECs has shown promise in restoring endothelial function, reducing vascular stiffness, and lowering blood pressure in hypertensive models. Understanding the complexity of EC heterogeneity and plasticity is critical for developing targeted therapies to prevent and treat cardiovascular diseases. By leveraging emerging genomic technologies and reprogramming strategies, future research may offer novel regenerative medicine approaches to restore vascular health and improve clinical outcomes for patients with cardiovascular diseases.
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