CX3CL1型                        
                
                                
                        
                            CX3CR1型                        
                
                                
                        
                            趋化因子                        
                
                                
                        
                            细胞生物学                        
                
                                
                        
                            生物                        
                
                                
                        
                            体内                        
                
                                
                        
                            绿色荧光蛋白                        
                
                                
                        
                            趋化因子受体                        
                
                                
                        
                            受体                        
                
                                
                        
                            分子生物学                        
                
                                
                        
                            生物化学                        
                
                                
                        
                            基因                        
                
                                
                        
                            遗传学                        
                
                        
                    
            作者
            
                Ki Wook Kim,Alexandra Vallon-Eberhard,Ehud Zigmond,Júlia Farache,Elias Shezen,Guy Shakhar,Andreas Ludwig,Sérgio A. Lira,Steffen Jung            
         
                    
            出处
            
                                    期刊:Blood
                                                         [Elsevier BV]
                                                        日期:2011-11-24
                                                        卷期号:118 (22): e156-e167
                                                        被引量:226
                                 
         
        
    
            
            标识
            
                                    DOI:10.1182/blood-2011-04-348946
                                    
                                
                                 
         
        
                
            摘要
            
            The CX3C chemokine family is composed of only one member, CX3CL1, also known as fractalkine, which in mice is the sole ligand of the G protein-coupled, 7-transmembrane receptor CX3CR1. Unlike classic small peptide chemokines, CX3CL1 is synthesized as a membrane-anchored protein that can promote integrin-independent adhesion. Subsequent cleavage by metalloproteases, either constitutive or induced, can generate shed CX3CL1 entities that potentially have chemoattractive activity. To study the CX3C interface in tissues of live animals, we generated transgenic mice (CX3CL1cherry:CX3CR1gfp), which express red and green fluorescent reporter genes under the respective control of the CX3CL1 and CX3CR1 promoters. Furthermore, we performed a structure/function analysis to differentiate the in vivo functions of membrane-tethered versus shed CX3CL1 moieties by comparing their respective ability to correct established defects in macrophage function and leukocyte survival in CX3CL1-deficient mice. Specifically, expression of CX3CL1(105Δ), an obligatory soluble CX3CL1 isoform, reconstituted the formation of transepithelial dendrites by intestinal macrophages but did not rescue circulating Ly6Clo CX3CR1hi blood monocytes in CX3CR1gfp/gfp mice. Instead, monocyte survival required the full-length membrane-anchored CX3CL1, suggesting differential activities of tethered and shed CX3CL1 entities.
         
            
 
                 
                
                    
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