安普克
血管紧张素II
内科学
肌肉肥大
内分泌学
蛋白激酶A
AMP活化蛋白激酶
磷酸化
心力衰竭
脱磷
磷酸酶
基因剔除小鼠
压力过载
细胞生物学
生物
化学
医学
受体
血压
心肌肥大
作者
Xue Han,Qiaojuan Shi,Yu Tu,Jiajia Zhang,Mengyang Wang,Weiqi Li,Y Liu,Ruyi Zheng,Jiajia Wei,Shiju Ye,Yanmei Zhang,Bozhi Ye,Junqing Wang,Huazhong Ying,Guang Liang
标识
DOI：10.1161/circresaha.124.325262
摘要
This study reveals that PRL2, as a novel AMPK-regulating phosphatase, promotes mitochondrial instability and hypertrophic injury in cardiomyocytes and provides PLR2 as a potential target for future drug development treating heart failure.
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