免疫系统
免疫学
医学
疾病
细胞因子
生物
免疫
国家（计算机科学）
抗体
免疫病理学
作者
Simone E. M. Olsthoorn,Anne Onrust-van Schoonhoven,Marjolein J. W. de Bruijn,Jochem Weekers,Menno van Nimwegen,Gregory van Beek,Willem de Koning,Lianne Trap,Esmee K. van der Ploeg,Geert H. van Weelden,Mathijs A. Sanders,Laura Surace,James P. Di Santo,Rudi W. Hendriks,Ralph Stadhouders
标识
DOI：10.1016/j.jaci.2026.05.029
摘要
BACKGROUND: ILC2 state remain poorly understood. OBJECTIVE: ILC2s and identified signals that induce this phenotype, including links with immune disease susceptibility. METHODS: ILC2s and multipotent ILC progenitors. Epigenomic data were integrated with genetic risk variants for major human immune diseases. RESULTS: ILC2s through STAT5 activation. CONCLUSIONS: ILC2s.
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