Targeting the PI3K/AKT/mTOR pathway in biliary tract cancers: A review of current evidences and future perspectives

PI3K/AKT/mTOR通路 蛋白激酶B 医学 PTEN公司 吉西他滨 癌症研究 癌变 肿瘤科 癌症 磷酸化 内科学 信号转导 生物 细胞生物学
作者
Francesca Corti,Federico Nichetti,Alessandra Raimondi,Monica Niger,Natalie Prinzi,Martina Torchio,Elena Tamborini,Federica Perrone,Giancarlo Pruneri,Maria Di Bartolomeo,Filippo de Braud,Sara Pusceddu
出处
期刊:Cancer Treatment Reviews [Elsevier BV]
卷期号:72: 45-55 被引量:99
标识
DOI:10.1016/j.ctrv.2018.11.001
摘要

Biliary tract cancers (BTCs) are a group of invasive neoplasms, with increasing incidence and dismal prognosis. In advanced disease, the standard of care is represented by first-line chemotherapy with cisplatin and gemcitabine. In subsequent lines, no clear recommendations are currently available, highlighting the need for novel therapeutic approaches. The PI3K/AKT/mTOR pathway is a core regulator of cell metabolism, growth and survival, and is involved in BTCs carcinogenesis and progression. Mutations, gene copy number alterations and aberrant protein phosphorylation of PI3K, AKT, mTOR and PTEN have been thoroughly described in BTCs and correlate with poor survival outcomes. Several pre-clinical evidences state the efficacy of PI3K/AKT/mTOR pathway inhibitors in BTCs, both in vitro and in vivo. In the clinical setting, initial studies with rapamycin analogs have shown interesting activity with an acceptable toxicity profile. Novel strategies evaluating AKT and PI3K inhibitors have risen serious safety concerns, pointing out the need for improved patient selection and increased target specificity for the clinical development of these agents, both alone and in combination with chemotherapy. This review extensively describes the role of the PI3K/AKT/mTOR pathway in BTCs and examines the rationale of its targeting in these tumors, with particular focus on clinical activity, toxicities and perspectives on further development of PI3K/AKT/mTOR pathway inhibitors.
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