体内
CD19
嵌合抗原受体
免疫疗法
癌症研究
癌症免疫疗法
离体
化学
抗原
癌症
淋巴瘤
体外
癌细胞
肿瘤抗原
表型
癌症治疗
受体
融合蛋白
癌症治疗
免疫系统
细胞疗法
遗传增强
作者
Y W Sun,Hong-Min Shang,Mei-Dan Wang,Meng-Wen Huang,Yu-Xin Qing,Yin-Yi Cai,Ying‐Li Luo,Zi-Dong Lu,Liu Bian-xia,Xiao‐Jiao Du,Song Shen,Xianzhu Yang,Jun Wang,Cong‐Fei Xu
出处
期刊:ACS Nano
[American Chemical Society]
日期:2026-07-04
卷期号:20 (27): 19163-19179
被引量:2
标识
DOI:10.1021/acsnano.5c21015
摘要
Chimeric antigen receptor macrophages (CAR-Ms) are promising in solid tumor therapy due to their tumor-penetrating property and antigen-specific phagocytosis. However, current CAR-M therapy is limited by the low ex vivo proliferation of macrophages and the complexity of the engineering process. Generating CAR-Ms in vivo can overcome these challenges but still faces an M2-like pro-tumor phenotype polarized by immunosuppressive tumor microenvironment. Herein, we devise macrophage-preferential ionizable cationic lipid-assisted polymeric nanoparticles (iCLANs) to co-deliver mRNAs encoding interferon-γ (IFN-γ) and a CAR molecule, denoted as iCLANmCAR+mIFN-γ, enabling in vivo engineering of CAR-Ms with a sustained M1-like phenotype. iCLANmCAR+mIFN-γ can coexpress IFN-γ and CAR in tumor-associated macrophages, thereby producing CAR-Ms capable of maintaining antitumor phenotype to effectively engulf tumor cells in an antigen-specific manner. Intravenous injection of iCLANmCAR+mIFN-γ in EGFRvIII+ breast tumor and CD19+ B-cell lymphoma models directly generates EGFRvIII CAR-Ms or CD19 CAR-Ms within tumors, resulting in significant tumor growth inhibition and remodeling of the immunosuppressive tumor microenvironment. This study provides an efficient strategy for in vivo engineering of M1-like CAR-Ms for cancer therapy.
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