发病机制
N6-甲基腺苷
甲基化
甲基转移酶
生物
生物信息学
机制(生物学)
单核苷酸多态性
基因
遗传学
免疫学
基因型
认识论
哲学
作者
Hongtao Chang,Yang Jun,Qianwen Wang,Jingjing Zhao,Ruixia Zhu
标识
DOI:10.1080/14737159.2022.2049246
摘要
Collectively, the information in the present review is a summary of the latest developments in m6A modification and highlights the mechanisms underlying IS pathogenesis, which may provide novel insights into the mechanisms and therapeutic targets for IS.
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