基因敲除
下调和上调
癌症研究
细胞生长
化学
背景（考古学）
活力测定
肝细胞癌
蛋白激酶B
细胞
生物
信号转导
细胞凋亡
生物化学
基因
古生物学
作者
Xiao Liu,Honglin Qu,Jingmin Li,Xuhong Sun,Zhenlin Wang,Dong Wang,Xianyong Bai,Xiaoyan Li
标识
DOI：10.3389/fonc.2025.1584811
摘要
These findings revealed that p53 enhances elesclomol-Cu-induced cuproptosis in HCC via FDXR-mediated FDX1 upregulation. This study provides mechanistic insights into p53's role in cuproptosis and may serve as a basis for targeting copper metabolism in therapeutic strategies for HCC.
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(2025-6-4)
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