癌症研究
生物
甲状腺癌
细胞生长
自噬
CXCR4型
转移
间质细胞
癌变
内分泌学
细胞凋亡
受体
甲状腺
癌症
趋化因子
生物化学
遗传学
作者
Hong Zhou,Xin Xie,Ying Chen,Yi Lin,Zhaogen Cai,Li Ding,Yijie Wu,Yongde Peng,Shanshan Tang,Huanbai Xu
标识
DOI:10.1210/clinem/dgaa366
摘要
Our investigation identified CMA regulated by ERα promoting PTC tumor progression that enhanced tumor cell proliferation and migration by targeting PPARγ-SDF1/CXCR4 signaling, representing a potential target for treatment of PTC.
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