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Highly expressed MT-ND3 positively associated with histological severity of hepatic steatosis

脂肪变性 脂毒性 脂肪肝 内科学 氧化应激 生物 内分泌学 脂质代谢 发病机制 细胞凋亡 病理 医学 生物化学 胰岛素抵抗 疾病 胰岛素
作者
Hanning Wang,Hongdong Chen,Kai‐Yun Chen,Jinfeng Xiao,Ke He,Guoan Xiang,Xin Xie
出处
期刊:Apmis [Wiley]
卷期号:122 (5): 443-451 被引量:7
标识
DOI:10.1111/apm.12166
摘要

Hepatic steatosis is the accumulation of an excess amount of triglycerides and other fats inside liver cells resulting from abnormal hepatic lipid metabolism. Mitochondrial structural and molecular defects are involved in the progression of hepatic steatosis pathogenesis. Hepatic methylation and transcriptional activity of the mitochondrial-encoded NADH dehydrogenase (MT-ND) play a critical role in the progression of non-alcoholic fatty liver disease (NAFLD). However, the expression of MT-ND3 in hepatic steatosis has not been extensively studied. In this study, liver specimens were collected from different patients, and were subjected to immunohistochemistry. Primary hepatocytes were treated with oxidative stress, hypoxia, and lipotoxicity to investigate the respective roles of these factors on MT-ND3 expression and cell apoptosis by western blotting and flow cytometry, respectively. We found that increased MT-ND3 expression in human hepatic steatosis was positively associated with histological severity of hepatic steatosis. Hypoxia, H2O2, and saturated fatty acid treatment induced cell apoptosis mediated by mitochondria. These three factors all had effects on MT-ND3 expression in cultured hepatocytes. Taken together, MT-ND3 may play important roles in hepatic steatosis progress. Hypoxia, oxidative stress, and lipotoxicity could all influence expression of MT-ND3 and thus may play a role in the progression of hepatic steatosis.
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