细胞毒性T细胞
细胞生物学
T细胞
生物
背景(考古学)
受体
细胞培养
细胞
分子生物学
体外
Jurkat细胞
免疫系统
功能(生物学)
化学
癌症研究
白细胞介素21
免疫学
体内
信号转导
肌动蛋白细胞骨架
抗原提呈细胞
T细胞受体
细胞毒性
ZAP70型
免疫疗法
受体表达
作者
Hanin Alamir,Carissa C. W. Wong,Amal Alsubaiti,Grace L. Edmunds,Maryam Alismail,L. Huynh,Yi-Wei Shi,Philip A. Lewis,Tressan Grant,Safaa Alsulaimani,J. Boyd,Christopher J. Holland,David J. Morgan,Awen Gallimore,Christoph Wülfing
出处
期刊:Science Signaling
[American Association for the Advancement of Science]
日期:2025-09-23
卷期号:18 (905): eadk4594-eadk4594
被引量:2
标识
DOI:10.1126/scisignal.adk4594
摘要
TIM3 is a coregulatory receptor that is highly abundant on multiple immune cell types, including T cells in response to prolonged exposure to antigen, and it marks functionally suppressed cytotoxic T lymphocytes (CTLs) in the tumor microenvironment. TIM3 exhibits inhibitory function in vivo but paradoxically has costimulatory T cell signaling capability in vitro. Here, we found that TIM3 directly inhibited the function of murine and human CTLs in direct interaction with target tumor cell spheroids. TIM3 regulated the ability of suppressed CTLs to polarize their actin cytoskeleton as a required step in cytolysis. Whereas the expression of the proposed TIM3 ligands CEACAM1 and galectin 9 in trans on target tumor cells enhanced TIM3 function, expression of CEACAM1 in cis on CTLs had the opposite effect. TIM3 functioned as an inhibitory receptor on spheroid-suppressed CTLs but not on active CTLs in a two-dimensional tissue culture model. Together, these data suggest that TIM3 enhances T cell function, serving as either a coinhibitory or costimulatory receptor depending on the functional context of the T cell on which it is expressed.
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