细胞生物学
计算生物学
生物
融合蛋白
融合
核孔蛋白
蛋白质-蛋白质相互作用
标识
DOI：10.1038/s41594-021-00558-w
摘要
NUP98 is one of the most promiscuous fusion partners involved in leukemogenic chromosomal translocations, but the myriad of partners has long obfuscated the mechanism by which these fusion proteins drive leukemia. A new mass spectrometry–based approach has produced clues that suggest an entirely new model of leukemogenesis.
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