Cholesterol metabolism: molecular mechanisms, biological functions, diseases, and therapeutic targets

计算生物学 前蛋白转化酶 胆汁酸 脂筏 胆固醇 细胞生物学 信号转导 生物 胆固醇逆向转运 PCSK9 代谢途径 肝X受体 生物信息学 焊剂(冶金) RNA干扰 细胞信号 化学 疾病 内化 脂肪肝 药理学 内体 葡萄糖稳态 生物化学 药物发现 NPC1 以兹提米比 细胞色素P450 受体 单酰甘油脂肪酶
作者
Daxin Cui,Xiaoqian Yu,Qiuyue Guan,Ying Shen,Jiajing Liao,Yin Liu,Zhiguang Su
出处
期刊:Molecular biomedicine [Springer Nature]
卷期号:6 (1): 72-72 被引量:28
标识
DOI:10.1186/s43556-025-00321-3
摘要

Cholesterol, an indispensable structural and signaling lipid, is fundamental to cellular membrane integrity, steroidogenesis, and developmental morphogen pathways. Its homeostasis hinges on the precise coordination of four interdependent metabolic modules: de novo biosynthesis, intestinal absorption, enzymatic conversion, and systemic clearance. This review delineates the molecular machinery governing these processes-from the Bloch/Kandutsch-Russell synthesis pathways and niemann-pick C1-like 1 (NPC1L1)-mediated cholesterol uptake to cholesterol 7α-hydroxylase (CYP7A1)-driven bile acid synthesis and HDL-dependent reverse transport. We further elucidate cholesterol's multifaceted roles in lipid raft assembly, Hedgehog signal transduction, and vitamin D/hormone production. Critically, dysregulation of cholesterol flux underpins pathogenesis in atherosclerosis, metabolic dysfunction-associated fatty liver disease (MAFLD), neurodegenerative disorders, and oncogenesis, with disrupted synthesis, efflux, or esterification cascades serving as key drivers. Emerging therapeutic strategies extend beyond conventional statins and proprotein convertase subtilisin/kexin type 9 (PCSK9) inhibitors to include transformative modalities: CRISPR-based in vivo gene editing (e.g., VERVE-101 targeting PCSK9), small interfering RNA (siRNA) therapeutics (inclisiran), and microbiota-directed interventions. Pioneering approaches against targets Such as angiopoietin-like 3 (ANGPTL3), lipoprotein(a) [Lp(a)], and asialoglycoprotein receptor 1 (ASGR1)-alongside repurposed natural agents (berberine, probiotics)-offer promise for mitigating residual cardiovascular risk and advancing precision cardiometabolic medicine. By integrating mechanistic insights with clinical advancements, this review underscores the transition from broad-spectrum therapies to personalized, multi-target regimens, offering a roadmap for mitigating cholesterol-related diseases in the era of genomic and metabolic medicine.
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