生物
串扰
发病机制
免疫系统
基因
特发性肺纤维化
线粒体
肺纤维化
纤维化
免疫功能障碍
遗传学
免疫学
生物信息学
肺
病理
内科学
医学
物理
光学
作者
Jun Huang,Xia Wang,Youjie Zeng,Huilin Xu,Siyi Zhang,Zhigang Ding,Ren Guo
出处
期刊:Gene
[Elsevier BV]
日期:2024-08-13
卷期号:930: 148840-148840
被引量:1
标识
DOI:10.1016/j.gene.2024.148840
摘要
Idiopathic pulmonary fibrosis (IPF) stands out as a life-threatening and one of the most severe interstitial lung diseases. The pathogenesis of IPF is not fully understood, while recent studies have highlighted the association of mitochondrial dysfunction with IPF. This study is dedicated to pinpointing crucial genes related to mitochondria that potentially impact the advancement of IPF, thereby offering new perspectives on the pathogenesis of this condition.
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