炎症体
炎症
医学
药理学
急性肾损伤
肾
细胞凋亡
上睑下垂
半胱氨酸蛋白酶
癌症研究
化学
内科学
程序性细胞死亡
生物化学
作者
Jianfei Weng,Dengyong Zheng,Huijun Chen,Zhangcheng Huang,Xiaojing Wu,Weijie Zheng,Zi Yu,Qinghui Xu
出处
期刊:Endocrine, metabolic & immune disorders
[Bentham Science Publishers]
日期:2025-07-14
卷期号:26: e18715303402744-e18715303402744
标识
DOI:10.2174/0118715303402744250704071034
摘要
This study demonstrates that YPSXZJDD alleviates HN-induced kidney injury by upregulating miR-223 and inhibiting the NLRP3/Caspase-1 pathway. The therapeutic potential of YPSXZJDD is supported by its ability to mitigate inflammation and renal damage, offering a promising approach for HN treatment. Further research into the broader role of miR-223 in kidney disease and related conditions is warranted to expand the understanding of its therapeutic applications.
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