自噬
线粒体
细胞生物学
内质网
机制(生物学)
氧化应激
冲程(发动机)
细胞凋亡
细胞内
平衡
粒体自噬
生物
神经科学
医学
生物信息学
生物化学
哲学
工程类
认识论
机械工程
作者
Li Lei,Shuaifeng Yang,Xiaoyang Lu,Yongfa Zhang,Tao Li
标识
DOI:10.3389/fnagi.2021.698601
摘要
Mitochondrial autophagy is an early defense and protection process that selectively clears dysfunctional or excessive mitochondria through a distinctive mechanism to maintain intracellular homeostasis. Mitochondrial dysfunction during cerebral stroke involves metabolic disbalance, oxidative stress, apoptosis, endoplasmic reticulum stress, and abnormal mitochondrial autophagy. This article reviews the research progress on the mechanism of mitochondrial autophagy in ischemic stroke to provide a theoretical basis for further research on mitochondrial autophagy and the treatment of ischemic stroke.
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