生物
外小体复合体
遗传学
核糖核酸
基因
转录组
错义突变
突变体
非编码RNA
突变
基因表达
作者
Maria C. Sterrett,Lauryn A. Cureton,Lauren N. Cohen,Ambro van Hoof,Sohail Khoshnevis,Milo B. Fasken,Anita H. Corbett,Homa Ghalei
出处
期刊:RNA
[Cold Spring Harbor Laboratory Press]
日期:2025-04-17
卷期号:31 (7): 988-1012
被引量:1
标识
DOI:10.1261/rna.080447.125
摘要
The RNA exosome is a multisubunit, evolutionarily conserved ribonuclease complex that is essential for processing, decay, and surveillance of many cellular RNAs. Missense mutations in genes encoding the structural subunits of the RNA exosome complex cause a diverse range of diseases, collectively known as RNA exosomopathies, often involving neurological and developmental defects. The varied symptoms suggest that different mutations lead to distinct in vivo consequences. To investigate these functional consequences and distinguish whether they are unique to each RNA exosomopathy mutation, we generated a collection of in vivo models by introducing pathogenic missense mutations in orthologous Saccharomyces cerevisiae genes. Comparative RNA-seq analysis assessing broad transcriptomic changes in each mutant model revealed that three yeast mutant models, rrp4-G226D , rrp40-W195R , and rrp46-L191H , which model mutations in the genes encoding EXOSC2, EXOSC3, and EXOSC5, respectively, had the largest transcriptomic differences. While some transcriptomic changes, particularly in transcripts related to ribosome biogenesis, were shared among mutant models, each mutation also induced unique transcriptomic changes. Thus, our data suggest that while there are some shared consequences, there are also distinct differences in RNA exosome function by each variant. Assessment of ribosome biogenesis and translation defects in the three models revealed distinct differences in polysome profiles. Collectively, our results provide the first comparative analyses of RNA exosomopathy mutant models and suggest that different RNA exosome gene mutations result in in vivo consequences that are both unique and shared across each variant, providing further insight into the biology underlying each distinct pathology.
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