醛糖还原酶
脂肪组织
衰老
内科学
内分泌学
脂肪细胞
脂解
醛还原酶
多元醇途径
肥胖
医学
生物
糖尿病
作者
Devi Thiagarajan,Nosirudeen Quadri,S Jawahar,Hylde Zirpoli,Carmen Hurtado del Pozo,Raquel López‐Díez,Syed Nurul Hasan,Gautham Yepuri,Paul F. Gugger,Brian S. Finlin,Philip A. Kern,Kenneth H. Gabbay,Ann Marie Schmidt,Ravichandran Ramasamy
出处
期刊:Obesity
[Wiley]
日期:2022-07-27
卷期号:30 (8): 1647-1658
被引量:12
摘要
AKR1B1/Akr1b3 modulation of senescence in subcutaneous adipose tissue contributes to aberrant metabolic responses to high-fat feeding. These data unveil new opportunities to target these pathways to combat obesity.
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