Redefining cell therapy: CAR-engineered innate immune cells to conquer solid and hematologic malignancies

先天免疫系统 嵌合抗原受体 生物 抗原 免疫系统 免疫学 归巢(生物学) 免疫疗法 癌症研究 计算生物学 先天性淋巴细胞 T细胞 模式识别受体 癌症免疫疗法 细胞 获得性免疫系统 癌症 干细胞 细胞毒性T细胞 细胞生物学 TLR9型 疾病 细胞毒性
作者
Ashik Anil Mathew,Ronak Raheja,K P Ramalingam
出处
期刊:Molecular Biology Reports [Springer Nature]
卷期号:53 (1): 254-254
标识
DOI:10.1007/s11033-025-11407-0
摘要

Chimeric antigen receptor (CAR) technology has revolutionized cancer therapy, yet its full potential remains untapped within the innate immune system. Beyond CAR-T cells, a growing cadre of MHC-independent effectors, including NK cells, macrophages, γδ T cells and the emerging innate-like T cells such as invariant NKT (iNKT) and mucosal-associated invariant T (MAIT) cells, offer complementary mechanisms for tumor recognition and elimination. These platforms combine facile, off-the-shelf manufacture from healthy donors with low graft-versus-host disease risk and a reduced propensity for severe cytokine release syndromes. Mechanistically, they span missing-self and antibody-dependent cytotoxicity (NK), phagocytosis and cross-presentation (macrophages), stress-ligand recognition (γδ T cells), and rapid, tissue-tropic, TCR-mediated responses to conserved lipid and metabolite antigens (iNKT via CD1d; MAIT via MR1). CAR engineering of these cells leverages their innate rapidity, innate/adaptive cross-talk, and distinctive homing to confront heterogeneous and immune-evasive tumors. Here, we synthesize recent advances in cell design, dual/split CARs, switchable control systems, armored payloads and synthetic-biology circuits, and evaluate translational progress, manufacturing bottlenecks, and regulatory considerations. We argue that integrating innate and innate-like programs with precision CAR architectures will yield a new generation of universal, resilient cellular therapeutics with broadened antigen reach, improved safety profiles, and enhanced capacity to overcome the suppressive tumor microenvironment.
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