Point-of-care tests of platelet reactivity and clot strength in risk assessment post-PCI: more insight into what really matters

Graphical AbstractOpen in new tabDownload slidePlatelet activation by exposure of plaque constituents and high shear forces, together with activation of coagulation, result in thrombus formation. This triggers simultaneous activation of endogenous fibrinolysis, and this, together with the effects of flow, may prevent vessel occlusion or restore vessel patency following thrombotic occlusion.