路易体
内嗅皮质
神经科学
神经退行性变
Tau病理学
α-突触核蛋白
皮质(解剖学)
混乱
τ蛋白
淀粉样蛋白(真菌学)
心理学
病理
医学
帕金森病
阿尔茨海默病
疾病
精神分析
海马体
作者
Fares Bassil,Hannah J. Brown,Shankar Pattabhiraman,Joe E. Iwasyk,Chantal Maghames,Emily S. Meymand,Timothy O. Cox,Dawn M. Riddle,Bin Zhang,John Q. Trojanowski,Virginia M.‐Y. Lee
出处
期刊:Neuron
[Elsevier]
日期:2023-11-01
卷期号:111 (22): 3699-3699
标识
DOI:10.1016/j.neuron.2023.10.030
摘要
(Neuron 105, 260–275.e1–e6; January 22, 2020) In the originally published article, the legend for Figure 1 contained an error, stating that panels G and H depicted tissue from frontal cortex. Instead, the legend should have stated that tissue was from entorhinal cortex. The original panels for Lewy body disease (LBD) in Figures 1B, 1G, and 1H were intentionally duplicated, as they all depicted the same condition. This was not explicitly mentioned in the text, however. To avoid further confusion, a corrected figure is provided here, displaying representative images of LBD in frontal cortex for Figures 1G and 1H. These changes do not affect any conclusions of the manuscript. We apologize for the error. Amyloid-Beta (Aβ) Plaques Promote Seeding and Spreading of Alpha-Synuclein and Tau in a Mouse Model of Lewy Body Disorders with Aβ PathologyBassil et al.NeuronNovember 20, 2019In BriefBassil et al. describe a new model that shows how Aβ plaques in the brain can potentiate α-synuclein pathogenesis. The authors provide evidence that the interaction of both proteins in the brain lead to neurodegeneration. Full-Text PDF Open Archive
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