纳米纤维
化学
伤口愈合
明胶
细胞生物学
活性氧
成纤维细胞
慢性伤口
血管生成
巨噬细胞
透明质酸
真皮成纤维细胞
细胞生长
静电纺丝
炎症
自愈水凝胶
细胞迁移
基因敲除
生物材料
细胞内
生物物理学
细胞
分泌物
新生血管
生物化学
信号转导
作者
Zihao Zou,Tianqi Zhou,Chengming Wen,Qiang Zou,Zeyang Yang,Wu Jieheng,Shunen Xu,Xu Ning,Chuan Ye,Wei Seong Toh,Long Yang
出处
期刊:ACS Nano
[American Chemical Society]
日期:2026-10-28
标识
DOI:10.1021/acsnano.6c14229
摘要
Abstract Chronic diabetic wounds remain difficult to treat because excessive reactive oxygen species (ROS), persistent inflammation, and impaired neovascularization collectively hinder tissue repair. Here, we present a bioinspired aligned electrospun nanofiber membrane composed of methacrylated gelatin and o-nitrobenzyl-modified gelatin and functionalized with manganese dioxide (MnO2) nanozymes to regulate the wound microenvironment. Upon hydration and brief ultraviolet irradiation, the membrane forms a conformable nanofibrous hydrogel that closely adapts to irregular wound beds. Among three MnO2 loadings, the formulation containing 180 μg/mL MnO2 was selected as the optimized formulation because it attenuated intracellular ROS while promoting fibroblast proliferation and migration and endothelial angiogenic activity. Mechanistic studies showed that the optimized hydrogel promoted fibroblast secretion of growth arrest-specific protein 6 (Gas6), which attenuated inflammatory macrophage responses and enhanced reparative macrophage responses. Gas6 knockdown weakened these effects, whereas recombinant Gas6 recapitulated them. Single-cell RNA sequencing identified treatment-associated changes in wound cell states and suggested Gas6-associated fibroblast-macrophage communication through computational ligand-receptor analysis. In streptozotocin-induced diabetic mice, the optimized hydrogel accelerated wound closure and enhanced vascularization, re-epithelialization, and collagen remodeling. These findings establish an aligned nanozyme-functionalized nanofibrous hydrogel that links redox regulation to Gas6-associated fibroblast-to-macrophage signaling for diabetic wound repair.
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