化学
活性氧
谷氨酰胺
氧化还原
肿瘤微环境
体内
生物物理学
氧化应激
药理学
肿瘤缺氧
癌症研究
谷胱甘肽
生物化学
缺氧(环境)
NADPH氧化酶
微泡
体外
细胞生物学
癌细胞
炎症
氧化磷酸化
肿瘤进展
癌症
过氧化氢酶
作者
Guanhua Qiu,Jie Long,Feng Lin,Ze‐Yan Yu,Renchuan Liang,Jiaxin Luo,XM Mo,Duo Wang,Lei Jiao,Yunxi Huang
标识
DOI:10.1186/s12951-025-03856-3
摘要
Therapeutic resistance remains a formidable challenge in oncology, mainly due to the chaotic tumor microenvironment (TME), which drive tumor progression and adaptive resistance via a self-sustaining metabolic-oxidative feedback loop. Herein, a multifunctional nanotherapeutic platform based on PdPtBi multimetallic nanozymes (PPB MMNs) is rationally engineered to address these challenges by simultaneously targeting reactive oxygen species (ROS) modulation and glutamine (Gln) metabolic disruption. Specifically, the PPB MMNs display cascade enzyme-mimicking activities resembling peroxidase (POD), catalase (CAT), and glutathione oxidase (GOD-SH). These activities effectively promote the generation of reactive oxygen species (ROS) and facilitate the decomposition of endogenous H₂O₂ into O₂, thereby alleviating hypoxia and inducing oxidative stress. Upon ultrasound (US) activation, their catalytic performance is further amplified, enabling deep-tissue therapeutic efficacy. Concurrently, PPB MMNs impair Gln metabolism, aggravating redox dyshomeostasis and undermining tumor metabolic flexibility. Both in vitro and in vivo studies validate the superior antitumor activity of PPB MMNs in suppressing primary and metastatic tumor growth by disrupting the redox-metabolic axis. Collectively, our study offers a robust framework for designing multimodal nanomedicines that reprogram the TME and overcome resistance, opening new avenues for precise, synergistic cancer therapy based on redox and metabolic vulnerabilities.
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