癌症研究
N6-甲基腺苷
医学
化学
生物化学
基因
甲基转移酶
甲基化
作者
Jiawei Qiu,Rongrong Zhao,Caizhi Ma,Qingtong Wang,Boyan Li,Yanhua Qi,Ziwen Pan,Shulin Zhao,Shaobo Wang,Zijie Gao,Xiaofan Guo,Wei Qiu,Weijie Tang,Xing Guo,Linhong Deng,Hao Xue,Gang Li
标识
DOI：10.1093/neuonc/noae268
摘要
Interactions between mesenchymal glioblastoma stem cells (MES GSCs) and myeloid-derived macrophages (MDMs) shape the tumor-immunosuppressive microenvironment (TIME), promoting the progression of glioblastoma (GBM). N6-methyladenosine (m6A) plays important roles in the tumor progression. However, the mechanism of m6A in shaping the TIME of GBM remains elusive.
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