心肌保护
缺血
金属硫蛋白
再灌注损伤
医学
心脏病学
心肌再灌注损伤
心肌缺血
内科学
药理学
化学
生物化学
基因
作者
Yoshio Oshima,Yasushi Fujio,Takashi Nakanishi,N ITOH,Yasunori Yamamoto,Shigeru Negoro,Katsuhiro Tanaka,Tadamitsu Kishimoto,Ichiro Kawase,Junichi Azuma
标识
DOI:10.1016/j.cardiores.2004.10.021
摘要
Activation of STAT3 protects myocardium from I/R injury in vivo. STAT3 mediates cardioprotection at least partially through MT1 and 2. STAT3 is a potential therapeutic target for I/R injury.
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