The IL-6/sIL-6R complex as a novel target for therapeutic approaches

糖蛋白130 细胞生物学 信号转导 生物 受体 信号 细胞内 免疫系统 融合蛋白 机制(生物学) 功能(生物学) 免疫学 车站3 基因 生物化学 认识论 哲学 重组DNA
作者
Stefan Rose‐John,Georg H. Waetzig,Jürgen Scheller,Joachim Grötzinger,Dirk Seegert
出处
期刊:Expert Opinion on Therapeutic Targets [Taylor & Francis]
卷期号:11 (5): 613-624 被引量:357
标识
DOI:10.1517/14728222.11.5.613
摘要

IL-6 plays a pivotal role in immune responses and certain oncologic conditions. The intense investigation of its biological activity and function led to the discovery of two different IL-6-driven signalling pathways. Binding to the membrane-bound IL-6 receptor (mIL-6R, CD126) causes the recruitment of two gp130 co-receptor molecules (CD130) and the activation of intracellular signalling cascades via gp130. Although this classical pathway is mainly limited to hepatocytes, neutrophils, monocytes/macrophages and certain other leukocyte populations, which express IL-6R on their surface, an alternative mechanism has also been described. Proteolytic cleavage of the mIL-6R protein or translation from alternatively spliced mRNA leads to the generation of a soluble form of the IL-6R (sIL-6R), which is likewise able to bind to IL-6. The resulting IL-6/sIL-6R complex is also capable of binding to gp130 and inducing intracellular signalling. Through this so-called 'trans-signalling' mechanism, IL-6 is able to stimulate cells that lack an endogenous mIL-6R. High levels of IL-6 and sIL-6R have been reported in several chronic inflammatory and autoimmune diseases as well as in cancer. Preclinical animal disease models have provided strong evidence that specific blockade of IL-6-regulated signalling pathways represents a promising approach for the therapy of these diseases. An optimised variant of the recently described fusion protein sgp30Fc is now heading towards its clinical evaluation.
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