脂肪变性
内科学
内分泌学
碳水化合物反应元件结合蛋白
脂肪肝
肝细胞
非酒精性脂肪肝
脂质代谢
葡萄糖稳态
基因剔除小鼠
生物
碳水化合物代谢
化学
胰岛素
胰岛素抵抗
医学
生物化学
基因
转录因子
受体
体外
疾病
作者
Ah‐Reum Oh,Yelin Jeong,Junjie Yu,Tam Dao,Jin Ku Kang,Young Hoon Jung,Seung‐Soon Im,Sang Bae Lee,Dongryeol Ryu,Utpal B. Pajvani,KyeongJin Kim
标识
DOI：10.1053/j.gastro.2022.11.019
摘要
Obesity predisposes to type 2 diabetes (T2D) and nonalcoholic fatty liver disease (NAFLD), but underlying mechanisms are incompletely understood. Potassium channel tetramerization domain-containing protein 17 (Kctd17) levels are increased in livers from obese mice and humans. In this study, we investigated the mechanism of increased Kctd17 and whether it is causal to obesity-induced metabolic complications.
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